{"id":11917,"date":"2025-06-17T07:01:46","date_gmt":"2025-06-17T05:01:46","guid":{"rendered":"https:\/\/valintermed.com\/?p=11917"},"modified":"2025-06-17T07:01:46","modified_gmt":"2025-06-17T05:01:46","slug":"sindrom-lennoksa-gasto-lgs","status":"publish","type":"post","link":"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/","title":{"rendered":"Lennox-Gastaut syndrome (LGS)"},"content":{"rendered":"<div class=\"fpm_start\"><\/div>\n<p>Lennox-Gastaut syndrome (LGS) is a severe form of childhood epilepsy characterized by a triad of diagnostic features: polymorphic seizures, diffuse slow bursts-slow waves on the electroencephalogram, and cognitive impairment. The disease belongs to the category of drug-resistant epileptic encephalopathies and usually manifests between the ages of 1 and 8 years, most often between 3 and 5 years of life. The pathology is characterized by a progressive course with the formation of stable resistance to anticonvulsant therapy and pronounced neurological dysfunction.<\/p>\n<div id=\"ez-toc-container\" class=\"ez-toc-v2_0_85 counter-flat ez-toc-counter ez-toc-light-blue ez-toc-container-direction\">\n<div class=\"ez-toc-title-container\">\n<p class=\"ez-toc-title\" style=\"cursor:inherit\">Content<\/p>\n<span class=\"ez-toc-title-toggle\"><a href=\"#\" class=\"ez-toc-pull-right ez-toc-btn ez-toc-btn-xs ez-toc-btn-default ez-toc-toggle\" aria-label=\"Toggle Table of Content\"><span class=\"ez-toc-js-icon-con\"><span class=\"\"><span class=\"eztoc-hide\" style=\"display:none;\">Toggle<\/span><span class=\"ez-toc-icon-toggle-span\"><svg style=\"fill: #999;color:#999\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\" class=\"list-377408\" width=\"20px\" height=\"20px\" viewbox=\"0 0 24 24\" fill=\"none\"><path d=\"M6 6H4v2h2V6zm14 0H8v2h12V6zM4 11h2v2H4v-2zm16 0H8v2h12v-2zM4 16h2v2H4v-2zm16 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href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%98%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%8F_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F_%D0%B8_%D0%B8%D0%BD%D1%82%D0%B5%D1%80%D0%B5%D1%81%D0%BD%D1%8B%D0%B5_%D0%B8%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B8%D0%B5_%D1%84%D0%B0%D0%BA%D1%82%D1%8B\" >History of the disease and interesting historical facts<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-2\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%AD%D0%BF%D0%B8%D0%B4%D0%B5%D0%BC%D0%B8%D0%BE%D0%BB%D0%BE%D0%B3%D0%B8%D1%8F\" >Epidemiology<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-3\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%93%D0%B5%D0%BD%D0%B5%D1%82%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B0%D1%8F_%D0%BF%D1%80%D0%B5%D0%B4%D1%80%D0%B0%D1%81%D0%BF%D0%BE%D0%BB%D0%BE%D0%B6%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D1%8C_%D0%BA_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%BC%D1%83_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8E\" >Genetic predisposition to this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-4\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%A4%D0%B0%D0%BA%D1%82%D0%BE%D1%80%D1%8B_%D1%80%D0%B8%D1%81%D0%BA%D0%B0_%D0%B2%D0%BE%D0%B7%D0%BD%D0%B8%D0%BA%D0%BD%D0%BE%D0%B2%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Risk factors for the development of this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-5\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%94%D0%B8%D0%B0%D0%B3%D0%BD%D0%BE%D1%81%D1%82%D0%B8%D0%BA%D0%B0_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Diagnosis of this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-6\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%9B%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D0%B5\" >Treatment<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-7\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%A1%D0%BF%D0%B8%D1%81%D0%BE%D0%BA_%D0%BB%D0%B5%D0%BA%D0%B0%D1%80%D1%81%D1%82%D0%B2_%D0%BF%D1%80%D0%B8%D0%BC%D0%B5%D0%BD%D1%8F%D0%B5%D0%BC%D1%8B%D1%85_%D0%B4%D0%BB%D1%8F_%D0%BB%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >List of drugs used to treat this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-8\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%9C%D0%BE%D0%BD%D0%B8%D1%82%D0%BE%D1%80%D0%B8%D0%BD%D0%B3_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Disease monitoring<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-9\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%92%D0%BE%D0%B7%D1%80%D0%B0%D1%81%D1%82%D0%BD%D1%8B%D0%B5_%D0%BE%D1%81%D0%BE%D0%B1%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D0%B8_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Age-related features of the disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-10\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%92%D0%BE%D0%BF%D1%80%D0%BE%D1%81%D1%8B_%D0%B8_%D0%BE%D1%82%D0%B2%D0%B5%D1%82%D1%8B\" >Questions and Answers<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-11\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/lennox-syndrome-gastro-lgs\/#%D0%A1%D0%BE%D0%B2%D0%B5%D1%82%D1%8B_%D0%BE%D1%82_%D0%B4%D0%BE%D0%BA%D1%82%D0%BE%D1%80%D0%B0_%D0%9E%D0%BB%D0%B5%D0%B3%D0%B0_%D0%9A%D0%BE%D1%80%D0%B6%D0%B8%D0%BA%D0%BE%D0%B2%D0%B0\" >Advice from Dr. Oleg Korzhikov<\/a><\/li><\/ul><\/nav><\/div>\n<h2><span class=\"ez-toc-section\" id=\"%D0%98%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%8F_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F_%D0%B8_%D0%B8%D0%BD%D1%82%D0%B5%D1%80%D0%B5%D1%81%D0%BD%D1%8B%D0%B5_%D0%B8%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B8%D0%B5_%D1%84%D0%B0%D0%BA%D1%82%D1%8B\"><\/span>History of the disease and interesting historical facts<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>The term &quot;Lennox-Gastaut syndrome&quot; was first introduced into medical practice in 1966 by American neurologists William G. Lennox and Jean Aicardi Gastaut. According to historical data, the first description of similar clinical manifestations was made in 1939 by Gibbs, who noted a specific EEG pattern in children with epilepsy. An interesting fact is that the disease was initially considered a variant of childhood absence epilepsy, but subsequent studies revealed its independent nosological form. In the 1970s, thanks to the development of neurophysiological diagnostic methods, characteristic EEG patterns were established, which made it possible to clearly differentiate LGS from other epileptic syndromes.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%AD%D0%BF%D0%B8%D0%B4%D0%B5%D0%BC%D0%B8%D0%BE%D0%BB%D0%BE%D0%B3%D0%B8%D1%8F\"><\/span>Epidemiology<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>According to modern epidemiological studies, the prevalence of Lennox-Gastaut syndrome is 0.1-0.2% among all forms of epilepsy and about 1-2% among childhood epileptic encephalopathies. Among patients with childhood epilepsy, the incidence of LGS varies between 0.5-1%. The incidence ratio between boys and girls is approximately 1.4:1. The highest risk of developing the syndrome is observed in children aged 1 to 5 years. According to a 2020 meta-analysis including data from 15 countries, the average annual incidence is 1.2 cases per 100,000 children.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%93%D0%B5%D0%BD%D0%B5%D1%82%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B0%D1%8F_%D0%BF%D1%80%D0%B5%D0%B4%D1%80%D0%B0%D1%81%D0%BF%D0%BE%D0%BB%D0%BE%D0%B6%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D1%8C_%D0%BA_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%BC%D1%83_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8E\"><\/span>Genetic predisposition to this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>The genetic component in the development of LGS is confirmed by numerous studies. The most significant are mutations in the following genes:<\/p>\n<ul>\n<li>SCN1A \u2013 encodes the \u03b11-subunit of the sodium channel<\/li>\n<li>GABRG2 \u2013 participates in the formation of GABA receptors<\/li>\n<li>STXBP1 \u2013 regulates the secretion of neurotransmitters<\/li>\n<li>CHD2 \u2013 plays a role in chromatin remodeling<\/li>\n<\/ul>\n<p>According to a study published in the journal Epilepsia (2019), \u201cpathogenic mutations are found in 30-40% of LGS patients.\u201d Particular attention is paid to new mutations that arise de novo, which account for up to 60% of all genetic disorders in this syndrome.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%A4%D0%B0%D0%BA%D1%82%D0%BE%D1%80%D1%8B_%D1%80%D0%B8%D1%81%D0%BA%D0%B0_%D0%B2%D0%BE%D0%B7%D0%BD%D0%B8%D0%BA%D0%BD%D0%BE%D0%B2%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Risk factors for the development of this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>The main risk factors for the development of LGS can be divided into several categories:<\/p>\n<ul>\n<li>Perinatal complications: hypoxic-ischemic damage to the central nervous system, intrauterine infections<\/li>\n<li>Cerebral pathologies: congenital anomalies of brain development, previous meningitis\/encephalitis<\/li>\n<li>Toxic effects: exposure to heavy metals, organic solvents<\/li>\n<li>Neurometabolic disorders: mitochondrial cytopathies, lysosomal storage diseases<\/li>\n<\/ul>\n<p>It is important to note that according to a study in the Journal of Child Neurology (2021), \u201cin 25% cases, the etiology cannot be determined even with a comprehensive examination.\u201d<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%94%D0%B8%D0%B0%D0%B3%D0%BD%D0%BE%D1%81%D1%82%D0%B8%D0%BA%D0%B0_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Diagnosis of this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>The diagnosis of LGS is based on a comprehensive assessment of clinical, electrophysiological and neuroimaging data. Key features:<\/p>\n<ul>\n<li>Polymorphic seizures: atonic, tonic-clonic, absences, focal<\/li>\n<li>Characteristic EEG pattern: slow bursts of 1.5-2.5 Hz<\/li>\n<li>Neuroimaging: MRI of the brain to rule out structural abnormalities<\/li>\n<li>Laboratory tests: blood chemistry, genetic testing<\/li>\n<\/ul>\n<p>Differential diagnosis includes other epileptic encephalopathies such as Dravet syndrome and myoclonic epilepsy.<\/p><script data-noptimize=\"\" data-wpfc-render=\"false\">\nfpm_start( \"true\" );\n<\/script>\n\n<h2><span class=\"ez-toc-section\" id=\"%D0%9B%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D0%B5\"><\/span>Treatment<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>The therapeutic strategy for LGS includes:<\/p>\n<ul>\n<li>Pharmacotherapy: valproate, lamotrigine, topiramate<\/li>\n<li>Ketogenic diet<\/li>\n<li>Surgical treatment: vagal stimulation, callosotomy<\/li>\n<li>Rehabilitation measures: neuropsychological correction<\/li>\n<\/ul>\n<p>It is important to note the limited effectiveness of standard antiepileptic therapy, which requires a search for alternative approaches.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%A1%D0%BF%D0%B8%D1%81%D0%BE%D0%BA_%D0%BB%D0%B5%D0%BA%D0%B0%D1%80%D1%81%D1%82%D0%B2_%D0%BF%D1%80%D0%B8%D0%BC%D0%B5%D0%BD%D1%8F%D0%B5%D0%BC%D1%8B%D1%85_%D0%B4%D0%BB%D1%8F_%D0%BB%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>List of drugs used to treat this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>The main drugs include:<\/p>\n<ul>\n<li>Valproic acid<\/li>\n<li>Lamotrigine<\/li>\n<li>Topiramate<\/li>\n<li>Rufinamide<\/li>\n<li>Tslobazam<\/li>\n<\/ul>\n<p>According to the ILAE recommendations (2020), the choice of drug depends on the type of attacks and individual tolerability.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%9C%D0%BE%D0%BD%D0%B8%D1%82%D0%BE%D1%80%D0%B8%D0%BD%D0%B3_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Disease monitoring<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Regular monitoring of patients with LGS includes:<\/p>\n<ul>\n<li>Monthly monitoring of seizure frequency<\/li>\n<li>Quarterly EEG monitoring<\/li>\n<li>Regular biochemical monitoring<\/li>\n<li>Cognitive assessment every 6 months<\/li>\n<\/ul>\n<p>The prognosis remains grave: according to statistics, \u201ccomplete seizure control is achieved in less than 10% patients.\u201d<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%92%D0%BE%D0%B7%D1%80%D0%B0%D1%81%D1%82%D0%BD%D1%8B%D0%B5_%D0%BE%D1%81%D0%BE%D0%B1%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D0%B8_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Age-related features of the disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Clinical manifestations of LGS vary depending on age:<\/p>\n<ul>\n<li>Preschool age: tonic seizures predominate<\/li>\n<li>School age: frequency of focal seizures increases<\/li>\n<li>Adolescence: development of drug resistance<\/li>\n<\/ul>\n<p>According to the Pediatric Neurology study (2022), \u201cearly onset of the disease (before 3 years) is associated with a more severe course.\u201d<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%92%D0%BE%D0%BF%D1%80%D0%BE%D1%81%D1%8B_%D0%B8_%D0%BE%D1%82%D0%B2%D0%B5%D1%82%D1%8B\"><\/span>Questions and Answers<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<ul>\n<li><strong>What tests are needed to confirm the diagnosis?<\/strong> To verify the diagnosis, a comprehensive examination is required, including EEG monitoring, MRI of the brain, genetic testing and biochemical blood analysis.<\/li>\n<li><strong>Is it possible to completely cure the disease?<\/strong> Unfortunately, complete recovery is possible only in isolated cases; the main goal of therapy is to control attacks and improve quality of life.<\/li>\n<li><strong>How often should you undergo examinations?<\/strong> Quarterly EEG and regular biochemical monitoring are recommended, especially when taking antiepileptic drugs.<\/li>\n<\/ul>\n<h2><span class=\"ez-toc-section\" id=\"%D0%A1%D0%BE%D0%B2%D0%B5%D1%82%D1%8B_%D0%BE%D1%82_%D0%B4%D0%BE%D0%BA%D1%82%D0%BE%D1%80%D0%B0_%D0%9E%D0%BB%D0%B5%D0%B3%D0%B0_%D0%9A%D0%BE%D1%80%D0%B6%D0%B8%D0%BA%D0%BE%D0%B2%D0%B0\"><\/span>Advice from Dr. Oleg Korzhikov<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>In my many years of practice, I often encounter questions from parents about the treatment of LGS. Here are the main recommendations:<\/p>\n<ul>\n<li>Start therapy as soon as possible after diagnosis.<\/li>\n<li>Do not stop the prescribed treatment without consulting your doctor.<\/li>\n<li>Follow all prescribed examinations regularly<\/li>\n<li>Pay special attention to rehabilitation measures<\/li>\n<\/ul>\n<p>\u201cIt is important to remember,\u201d emphasizes Dr. Korzhikov, \u201cthat timely diagnosis and properly selected therapy can significantly improve the prognosis of the disease.\u201d<\/p>\n<div class=\"fpm_end\"><\/div>","protected":false},"excerpt":{"rendered":"<p>Lennox-Gastaut syndrome (LGS) is a severe form of childhood epilepsy characterized by a triad of diagnostic features: polymorphic seizures, diffuse slow bursts-slow waves on<\/p>","protected":false},"author":1,"featured_media":20461,"comment_status":"open","ping_status":"","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[298],"tags":[],"class_list":["post-11917","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-medlibrary"],"_links":{"self":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts\/11917","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/comments?post=11917"}],"version-history":[{"count":1,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts\/11917\/revisions"}],"predecessor-version":[{"id":15281,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts\/11917\/revisions\/15281"}],"wp:featuredmedia":[{"embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/media\/20461"}],"wp:attachment":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/media?parent=11917"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/categories?post=11917"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/tags?post=11917"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}