{"id":11208,"date":"2025-07-27T06:27:37","date_gmt":"2025-07-27T04:27:37","guid":{"rendered":"https:\/\/valintermed.com\/?p=11208"},"modified":"2025-07-27T06:27:37","modified_gmt":"2025-07-27T04:27:37","slug":"semeynaya-gipokaltsiuricheskaya-giperkaltsiemiya-tip-1","status":"publish","type":"post","link":"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/","title":{"rendered":"Familial hypocalciuric hypercalcemia type 1"},"content":{"rendered":"<div class=\"fpm_start\"><\/div>\n<p>Familial hypocalciuric hypercalcemia (FHCH) is a genetic disorder characterized by elevated blood calcium levels (hypercalcemia) and low urinary calcium excretion (hypocalciuria). This condition is usually caused by dysfunction of the parathyroid glands, which are responsible for regulating calcium metabolism in the body. FHCH is most often inherited in an autosomal dominant pattern. Due to changes in the body&#039;s sensitivity to parathyroid hormone (PTH) and other factors associated with calcium metabolism, clinical manifestations of this condition may develop in the form of fatigue, muscle weakness, increased thirst, and type 2 diabetes. Note that this disease requires careful diagnosis and treatment to avoid potentially serious complications.<\/p>\n<div id=\"ez-toc-container\" class=\"ez-toc-v2_0_86 counter-flat ez-toc-counter ez-toc-light-blue ez-toc-container-direction\">\n<div class=\"ez-toc-title-container\">\n<p class=\"ez-toc-title\" style=\"cursor:inherit\">Content<\/p>\n<span class=\"ez-toc-title-toggle\"><a href=\"#\" class=\"ez-toc-pull-right ez-toc-btn ez-toc-btn-xs ez-toc-btn-default ez-toc-toggle\" aria-label=\"Toggle Table of Content\"><span class=\"ez-toc-js-icon-con\"><span class=\"\"><span class=\"eztoc-hide\" style=\"display:none;\">Toggle<\/span><span class=\"ez-toc-icon-toggle-span\"><svg style=\"fill: #999;color:#999\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\" class=\"list-377408\" width=\"20px\" height=\"20px\" viewbox=\"0 0 24 24\" fill=\"none\"><path d=\"M6 6H4v2h2V6zm14 0H8v2h12V6zM4 11h2v2H4v-2zm16 0H8v2h12v-2zM4 16h2v2H4v-2zm16 0H8v2h12v-2z\" fill=\"currentColor\"><\/path><\/svg><svg 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href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%98%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%8F_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F_%D0%B8_%D0%B8%D0%BD%D1%82%D0%B5%D1%80%D0%B5%D1%81%D0%BD%D1%8B%D0%B5_%D0%B8%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B8%D0%B5_%D1%84%D0%B0%D0%BA%D1%82%D1%8B\" >History of the disease and interesting historical facts<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-2\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%AD%D0%BF%D0%B8%D0%B4%D0%B5%D0%BC%D0%B8%D0%BE%D0%BB%D0%BE%D0%B3%D0%B8%D1%8F\" >Epidemiology<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-3\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%93%D0%B5%D0%BD%D0%B5%D1%82%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B0%D1%8F_%D0%BF%D1%80%D0%B5%D0%B4%D1%80%D0%B0%D1%81%D0%BF%D0%BE%D0%BB%D0%BE%D0%B6%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D1%8C_%D0%BA_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%BC%D1%83_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8E\" >Genetic predisposition to this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-4\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%A4%D0%B0%D0%BA%D1%82%D0%BE%D1%80%D1%8B_%D1%80%D0%B8%D1%81%D0%BA%D0%B0_%D0%B2%D0%BE%D0%B7%D0%BD%D0%B8%D0%BA%D0%BD%D0%BE%D0%B2%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Risk factors for the development of this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-5\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%94%D0%B8%D0%B0%D0%B3%D0%BD%D0%BE%D1%81%D1%82%D0%B8%D0%BA%D0%B0_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Diagnosis of this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-6\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%9B%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D0%B5\" >Treatment<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-7\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%A1%D0%BF%D0%B8%D1%81%D0%BE%D0%BA_%D0%BB%D0%B5%D0%BA%D0%B0%D1%80%D1%81%D1%82%D0%B2_%D0%BF%D1%80%D0%B8%D0%BC%D0%B5%D0%BD%D1%8F%D0%B5%D0%BC%D1%8B%D1%85_%D0%B4%D0%BB%D1%8F_%D0%BB%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >List of medications used to treat this disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-8\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%9C%D0%BE%D0%BD%D0%B8%D1%82%D0%BE%D1%80%D0%B8%D0%BD%D0%B3_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Disease monitoring<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-9\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%92%D0%BE%D0%B7%D1%80%D0%B0%D1%81%D1%82%D0%BD%D1%8B%D0%B5_%D0%BE%D1%81%D0%BE%D0%B1%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D0%B8_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\" >Age-related features of the disease<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-10\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%92%D0%BE%D0%BF%D1%80%D0%BE%D1%81%D1%8B_%D0%B8_%D0%BE%D1%82%D0%B2%D0%B5%D1%82%D1%8B\" >Questions and Answers<\/a><\/li><li class='ez-toc-page-1'><a class=\"ez-toc-link ez-toc-heading-11\" href=\"https:\/\/valintermed.com\/en\/medlibrary\/family-hypokaltsiuricheskaya-hyperkaltsiemiya-type-1\/#%D0%A1%D0%BE%D0%B2%D0%B5%D1%82%D1%8B_%D0%BE%D1%82_%D0%B4%D0%BE%D0%BA%D1%82%D0%BE%D1%80%D0%B0_%D0%9E%D0%BB%D0%B5%D0%B3%D0%B0_%D0%9A%D0%BE%D1%80%D0%B6%D0%B8%D0%BA%D0%BE%D0%B2%D0%B0\" >Advice from Dr. Oleg Korzhikov<\/a><\/li><\/ul><\/nav><\/div>\n<h2><span class=\"ez-toc-section\" id=\"%D0%98%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%8F_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F_%D0%B8_%D0%B8%D0%BD%D1%82%D0%B5%D1%80%D0%B5%D1%81%D0%BD%D1%8B%D0%B5_%D0%B8%D1%81%D1%82%D0%BE%D1%80%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B8%D0%B5_%D1%84%D0%B0%D0%BA%D1%82%D1%8B\"><\/span>History of the disease and interesting historical facts<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Familial hypocalciuric hypercalcemia was first described in the medical literature in the 1960s. Research at that time showed that the disorder of calcium metabolism can be hereditary. In 1996, it was discovered that hypocalciuric hypercalcemia is caused by a mutation in the CASR gene, which is responsible for encoding the calcium receptor. In the following decades, studies were conducted confirming the presence of various mutations of this gene in patients diagnosed with FHH. A number of cases were also recorded where this disease was observed in several members of the same family, which confirmed its hereditary nature. <\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%AD%D0%BF%D0%B8%D0%B4%D0%B5%D0%BC%D0%B8%D0%BE%D0%BB%D0%BE%D0%B3%D0%B8%D1%8F\"><\/span>Epidemiology<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Familial hypocalciuric hypercalcemia is a rare disorder, but accurate statistics on its prevalence are limited. It is estimated to affect 1 to 4 people per 100,000 population, which creates the impression that there is a lack of relevant data to understand the scale of the problem. The disease can manifest itself at any age, but is most often diagnosed in young people or adults. Due to the autosomal dominant type of inheritance, the pathology is observed in both men and women equally.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%93%D0%B5%D0%BD%D0%B5%D1%82%D0%B8%D1%87%D0%B5%D1%81%D0%BA%D0%B0%D1%8F_%D0%BF%D1%80%D0%B5%D0%B4%D1%80%D0%B0%D1%81%D0%BF%D0%BE%D0%BB%D0%BE%D0%B6%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D1%8C_%D0%BA_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%BC%D1%83_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8E\"><\/span>Genetic predisposition to this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Familial hypocalciuric hypercalcemia is associated with mutations in the CASR gene, which codes for calcium receptors located on the surface of cells. This mutation leads to a decrease in the sensitivity of these cells to calcium, which in turn causes hypercalcemia, since the parathyroid glands continue to produce parathyroid hormone despite the discrepancy in calcium levels in the blood. In addition to CASR, genes involved may include GNA11 and AP2S1, which are also involved in the regulation of calcium metabolism. Inheritance of the disease occurs in an autosomal dominant manner, which means that one mutation in the gene of one parent is enough for the manifestation of symptoms of the disease in a child.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%A4%D0%B0%D0%BA%D1%82%D0%BE%D1%80%D1%8B_%D1%80%D0%B8%D1%81%D0%BA%D0%B0_%D0%B2%D0%BE%D0%B7%D0%BD%D0%B8%D0%BA%D0%BD%D0%BE%D0%B2%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Risk factors for the development of this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Familial hypocalciuric hypercalcemia is primarily determined by genetic factors, but there are risk factors that may contribute to the development of the disease. These include:<\/p>\n<ul>\n<li>Heredity: presence of first-degree relatives with the disease.<\/li>\n<li>Age: Symptoms are more often observed in patients under 30 years of age.<\/li>\n<li>Genetic mutations: presence of specific mutations in genes such as CASR.<\/li>\n<li>Consumption of foods high in calcium: may increase hypercalcemia in susceptible patients.<\/li>\n<\/ul>\n<p>Each of these factors can have a different impact on how quickly symptoms develop and the severity of the disease.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%94%D0%B8%D0%B0%D0%B3%D0%BD%D0%BE%D1%81%D1%82%D0%B8%D0%BA%D0%B0_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Diagnosis of this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Diagnosis of familial hypocalciuric hypercalcemia includes several stages:<\/p>\n<ul>\n<li>Main symptoms: Patients usually complain of fatigue, muscle weakness, increased thirst and frequent urination.<\/li>\n<li>Laboratory tests: detection of hypercalcemia (serum calcium level above 10.5 mg\/dl), hypocalciuria (urinary calcium excretion less than 100 mg\/day).<\/li>\n<li>Radiological tests: may help rule out other pathologies such as osteoporosis or tumor.<\/li>\n<li>Other types of diagnostics: molecular genetic testing to identify mutations in genes associated with the disease.<\/li>\n<li>Differential diagnosis: it is important to exclude primary hyperparathyroidism, sarcoidosis, malignant neoplasms with secondary hypercalcemia.<\/li>\n<\/ul>\n<p>Correct diagnosis plays a critical role in choosing effective treatment.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%9B%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D0%B5\"><\/span>Treatment<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Treatment of familial hypocalciuric hypercalcemia should be comprehensive:<\/p><script data-noptimize=\"\" data-wpfc-render=\"false\">\nfpm_start( \"true\" );\n<\/script>\n\n<ul>\n<li>General treatment: Lifestyle changes, including a diet restricted in calcium and phosphorus, require an individual approach.<\/li>\n<li>Pharmacological treatment: In some cases, calcitonin and bisphosphonates may be used to control calcium levels.<\/li>\n<li>Surgical treatment: indicated in the presence of other diseases of the parathyroid glands, such as adenoma or hyperplasia.<\/li>\n<li>Other treatments include drinking moderate amounts of fluids to lower the calcium levels in the blood.<\/li>\n<\/ul>\n<p>It is important that the treatment is regulated by an experienced endocrinologist with post-laboratory control.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%A1%D0%BF%D0%B8%D1%81%D0%BE%D0%BA_%D0%BB%D0%B5%D0%BA%D0%B0%D1%80%D1%81%D1%82%D0%B2_%D0%BF%D1%80%D0%B8%D0%BC%D0%B5%D0%BD%D1%8F%D0%B5%D0%BC%D1%8B%D1%85_%D0%B4%D0%BB%D1%8F_%D0%BB%D0%B5%D1%87%D0%B5%D0%BD%D0%B8%D1%8F_%D0%B4%D0%B0%D0%BD%D0%BD%D0%BE%D0%B3%D0%BE_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>List of medications used to treat this disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>The following medications may be used to treat familial hypocalciuric hypercalcemia:<\/p>\n<ul>\n<li>Calcitonin.<\/li>\n<li>Bisphosphonates (eg, alendronate).<\/li>\n<li>Hormones that regulate calcium metabolism (for example, parathyroid hormone). <\/li>\n<li>Diuretics (thiazides) in some cases - to control calcium levels.<\/li>\n<li>Vitamin D restriction if necessary.<\/li>\n<\/ul>\n<p>The appointment of appropriate therapy should only be carried out under the supervision of a specialist.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%9C%D0%BE%D0%BD%D0%B8%D1%82%D0%BE%D1%80%D0%B8%D0%BD%D0%B3_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Disease monitoring<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Monitoring the condition of a patient with familial hypocalciuric hypercalcemia requires regular observation:<\/p>\n<ul>\n<li>Control stages: tests for calcium levels in the blood and urine, assessment of kidney function, and the condition of the parathyroid glands.<\/li>\n<li>Prognosis: With timely and adequate therapy, the prognosis can be favorable. However, ignoring \u75c7\u72b6 can lead to complications such as osteoporosis, kidney stones, and others.<\/li>\n<li>Complications: may arise as a consequence of the disease itself or as a result of incorrectly selected treatment.<\/li>\n<\/ul>\n<p>Regular medical examinations significantly reduce the risk of complications.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%92%D0%BE%D0%B7%D1%80%D0%B0%D1%81%D1%82%D0%BD%D1%8B%D0%B5_%D0%BE%D1%81%D0%BE%D0%B1%D0%B5%D0%BD%D0%BD%D0%BE%D1%81%D1%82%D0%B8_%D0%B7%D0%B0%D0%B1%D0%BE%D0%BB%D0%B5%D0%B2%D0%B0%D0%BD%D0%B8%D1%8F\"><\/span>Age-related features of the disease<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Familial hypocalciuric hypercalcemia can occur in different age groups, but susceptibility to the disease and the severity of symptoms may vary:<\/p>\n<ul>\n<li>In children: often diagnosed at an early age, may have minor symptoms.<\/li>\n<li>In young people: symptoms often become more pronounced, phases of hypercalcemia may occur.<\/li>\n<li>In older people: symptoms may be less severe, but the risk of complications is significantly higher.<\/li>\n<\/ul>\n<p>Depending on age, treatment approaches and prescriptions may vary.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"%D0%92%D0%BE%D0%BF%D1%80%D0%BE%D1%81%D1%8B_%D0%B8_%D0%BE%D1%82%D0%B2%D0%B5%D1%82%D1%8B\"><\/span>Questions and Answers<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<ul>\n<li><strong>What causes familial hypocalciuric hypercalcemia?<\/strong><br \/>\nThe cause is mutations in genes associated with the regulation of calcium metabolism, most often in CASR.<\/li>\n<li><strong>What are the main symptoms of this disease?<\/strong><br \/>\nThe main symptoms include fatigue, muscle weakness, increased thirst and frequent urination.<\/li>\n<li><strong>How to diagnose hypercalcemia?<\/strong><br \/>\nDiagnosis is made through laboratory tests, including serum calcium levels and urine analysis.<\/li>\n<li><strong>How is familial hypocalciuric hypercalcemia treated?<\/strong><br \/>\nTreatment includes lifestyle changes, drug therapy, and, in rare cases, surgery.<\/li>\n<li><strong>What is the prognosis for patients with this disease?<\/strong><br \/>\nThe prognosis with adequate therapy is positive, but regular monitoring is necessary to prevent complications.<\/li>\n<\/ul>\n<h2><span class=\"ez-toc-section\" id=\"%D0%A1%D0%BE%D0%B2%D0%B5%D1%82%D1%8B_%D0%BE%D1%82_%D0%B4%D0%BE%D0%BA%D1%82%D0%BE%D1%80%D0%B0_%D0%9E%D0%BB%D0%B5%D0%B3%D0%B0_%D0%9A%D0%BE%D1%80%D0%B6%D0%B8%D0%BA%D0%BE%D0%B2%D0%B0\"><\/span>Advice from Dr. Oleg Korzhikov<span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>If you have familial hypocalciuric hypercalcemia, it is important to monitor your lifestyle and undergo regular check-ups. I recommend monitoring your calcium levels and paying attention to any changes in your well-being. You also need to pay attention to your diet, excluding excessive consumption of calcium-rich foods. And, of course, follow the treatment prescribed by your doctor and do not ignore the prescribed control tests. Discuss all issues of interest to your doctor to avoid serious complications.<\/p>\n<div class=\"fpm_end\"><\/div>","protected":false},"excerpt":{"rendered":"<p>Familial hypocalciuric hypercalcemia (FHCH) is a genetic disorder characterized by elevated blood calcium levels (hypercalcemia) in the presence of low calcium excretion.<\/p>","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[298],"tags":[],"class_list":["post-11208","post","type-post","status-publish","format-standard","hentry","category-medlibrary"],"_links":{"self":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts\/11208","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/comments?post=11208"}],"version-history":[{"count":1,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts\/11208\/revisions"}],"predecessor-version":[{"id":16007,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/posts\/11208\/revisions\/16007"}],"wp:attachment":[{"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/media?parent=11208"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/categories?post=11208"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/valintermed.com\/en\/wp-json\/wp\/v2\/tags?post=11208"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}