{"id":11049,"date":"2026-03-02T22:01:57","date_gmt":"2026-03-02T21:01:57","guid":{"rendered":"https:\/\/valintermed.com\/?p=11049"},"modified":"2026-03-02T22:01:57","modified_gmt":"2026-03-02T21:01:57","slug":"defekt-endokardialnoy-podushki","status":"publish","type":"post","link":"https:\/\/valintermed.com\/en\/medlibrary\/defect-of-the-endocardial-cushion\/","title":{"rendered":"Endocardial cushion defect"},"content":{"rendered":"
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The defect of the endocardial cushion is a congenital heart defect in which the normal formation of structures separating the right and left parts of the heart by the interatrial and interventricular septa is disrupted. It is not just a \"hole\" in the heart, but a complex developmental anomaly affecting several key elements: the atrioventricular valves (mitral and tricuspid), the septa, and the walls of the ventricles. As a result, a mixed blood shunt occurs \u2014 part of the blood from the left atrium enters the right, and from the left ventricle \u2014 into the right, leading to overload of the pulmonary circulation, hypertension in the pulmonary artery, and ultimately heart failure. In newborns, this defect may manifest within the first days of life: shortness of breath, cyanosis, poor weight gain, rapid heartbeat. However, symptoms may also appear later \u2014 in early childhood or even in adulthood, especially if the defect is partial and functional disturbances are compensated by the body.<\/p>\n

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